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Hepatic steatosis is defined as intrahepatic fat of at least 5% of liver weight. Simple accumulation of triacylglycerols in the liver could be hepatoprotective; however, prolonged hepatic lipid storage may lead to liver metabolic dysfunction, inflammation, and advanced forms of nonalcoholic fatty liver disease. Nonalcoholic hepatic steatosis is associated with obesity, type 2 diabetes, and dyslipidemia. Several mechanisms are involved in the accumulation of intrahepatic fat, including increased flux of fatty acids to the liver, increased de novo lipogenesis, and/or reduced clearance through β-oxidation or very-low-density lipoprotein secretion. This article summarizes the mechanisms involved in the accumulation of triacylglycerols in the liver, the clinical implications, and the prevention of hepatic steatosis, with a focus on the role of mitochondrial function and lifestyle modifications.
Research Article: Journal of Infectious Diseases & Preventive Medicine
Research Article: Journal of Infectious Diseases & Preventive Medicine
Research Article: Journal of Infectious Diseases & Preventive Medicine
Research Article: Journal of Infectious Diseases & Preventive Medicine
Research Article: Journal of Infectious Diseases & Preventive Medicine
Commentary: Journal of Infectious Diseases & Preventive Medicine
Keynote: Journal of Clinical Trials
Scientific Tracks Abstracts: Clinical & Experimental Cardiology
Keynote: Clinical & Experimental Cardiology
Posters & Accepted Abstracts: Clinical & Experimental Cardiology
Accepted Abstracts: Journal of Nutrition & Food Sciences
Scientific Tracks Abstracts: Journal of Nutrition & Food Sciences
Scientific Tracks Abstracts: Journal of Clinical & Experimental Dermatology Research