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Journal of Clinical & Experimental Dermatology Research

Journal of Clinical & Experimental Dermatology Research
Open Access

ISSN: 2155-9554

+44 1478 350008

Journal of Clinical & Experimental Dermatology Research : Citations & Metrics Report

Articles published in Journal of Clinical & Experimental Dermatology Research have been cited by esteemed scholars and scientists all around the world. Journal of Clinical & Experimental Dermatology Research has got h-index 8, which means every article in Journal of Clinical & Experimental Dermatology Research has got 8 average citations.

Following are the list of articles that have cited the articles published in Journal of Clinical & Experimental Dermatology Research.

  2022 2021 2020 2019 2018 0 0

Year wise published articles

30 59 57 40 37 0 0

Year wise citations received

182 207 222 227 168 140 21
Journal total citations count 278
Journal impact factor 0.98
Journal 5 years impact factor 0.7
Journal cite score 0.57
Journal h-index 8
Journal h-index since 2018 7
Important citations (413)

melnik bc, john sm, carrera-bastos p, cordain l. hairlosstalk forums.

melnik bc, john sm, carrera-bastos p, cordain l. hairlosstalk forums.

stage i. hidradenitis suppurativa relatively common.

poster a, melnik bc, john sm, carrera-bastos p, cordain l. hair loss help» hair loss open topic» dairy products (milk) & hair androgen receptor relationship.

melnik bc (2014) acne and genetics. in pathogenesis and treatment of acne and rosacea. springer berlin heidelberg pp: 109-130.

bowe wp, glick jb, shalita ar (2012) solodyn and updates on topical and oral therapies for acne. curr dermatol rep 1: 97-107.

danby fw (2011) acne: diet and acnegenesis. indian dermatol online j 2: 2.

gill l, williams m, hamzavi i (2014) update on hidradenitis suppurativa: connecting the tracts. f1000prime reports 6.

szabó k, kemény l (2011) studying the genetic predisposing factors in the pathogenesis of acne vulgaris. human immunol 72: 766-773.

melnik b (2011) isotretinoin and foxo1: a scientific hypothesis. dermato-endocrinol 3: 141-165.

arora mk, yadav a, saini v (2011) role of hormones in acne vulgaris. clin biochem 44: 1035-1040.

al-hassnawi at, al-ameria am, al-hassnawib hh (2015) association between antistreptolysin-o serum level and chronic plaque psoriasis in iraqi patients. j contemp med sci 1: 20-22.

yazici c, köse k, utaÅŸ s, tanrikulu e, taÅŸlidere n (308) a novel approach in psoriasis: first usage of known protein oxidation markers to prove oxidative stress. arch dermatol res 308: 207-212.

marrot l (2016) the cutaneous melanocyte as a target of environmental stressors: molecular mechanisms and opportunities. inskin stress response pathways: 175-196.

lerche cm, philipsen pa, poulsen t, gniadecki r, wulf hc (2012) topical nutlin‐3a does not decrease photocarcinogenesis induced by simulated solar radiation in hairless mice. photodermatol photoimmunol photomed 28: 207-212.

arun kumar r, sathish kumar d, gayatri v, nishanth t (2011) a comprehensive assessment and perception of genetically modified foods. j genetic syndromes gene ther.

alikhan a, ibrahimi oa, eisen db (2012) congenital melanocytic nevi: where are we now?: part i. clinical presentation, epidemiology, pathogenesis, histology, malignant transformation, and neurocutaneous melanosis. j am acad dermatol 67: 495-e1.

chauhan ps, vikram k mahajan, gayatri khatri, pushpinder s chauhan, karaninder s mehta, et al. (2015) progressive symmetric erythrokeratoderma having overlapping features with erythrokeratoderma variabilis and lesional hypertrichosis: is nomenclature “erythrokeratoderma variabilis progressiva” more appropriate? indian j dermatol 60: 410-411.

mahajan vk, khatri g, chauhan ps, mehta ks, raina r, et al. (2015) progressive symmetric erythrokeratoderma having overlapping features with erythrokeratoderma variabilis and lesional hypertrichosis: is nomenclature “erythrokeratoderma variabilis progressiva” more appropriate?. indian j dermatol 60: 410.

oliveira lc. estudo de associação entre polimorfismos e nivéis séricos do receptor 1 do complemento e o penfigo foláceo.

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